Expert Panel Highlights Serious Public Health Threats from Industrial Animal Agriculture

SAVE THE SACRED SITES ALLIANCE

http://www.pewtrusts.org/news_room_detail.aspx?id=37968

Contact: Ralph Loglisci, 202.223.2996

Washington, DC - 04/11/2008 - The same techniques that have increased the productivity of modern animal agriculture are also contributing to a number of growing public health concerns, a panel of experts told Congress today.

Members of the Pew Commission on Industrial Farm Animal Production (PCIFAP) enumerated the hazards to human health associated with today’s large-scale industrial farm animal production (IFAP). These hazards include exposure to harmful contaminants, the spread of infectious diseases, and a growing resistance to the antibiotics commonly used to treat those diseases.

Commissioners explained that some of the dominant practices used in IFAP facilities set the stage for these threats to emerge. In IFAP systems, large numbers of animals are raised together, usually in confinement buildings, increasing human exposure to dangerous pathogens from animals or the large quantity of animal wastes generated in such conditions. Animal waste, which harbors a number of pathogens and chemical contaminants, is usually left untreated, often sprayed on fields as fertilizer, raising the potential for contamination of air, water, and soils. In one recent example, farm animal run-off from IFAP facilities was among the suspected causes of a 2006 E. coli outbreak in which six people died and more than 250 were sickened.

There are numerous known “zoonotic” diseases -- infectious diseases that can be transmitted between humans and animals. Due to the large numbers of animals housed in close quarters in typical IFAP facilities, there are many opportunities for animals to be infected by several strains of pathogens, leading to increased chance for a strain to emerge that can infect and spread in humans.

Another concern stems from the use of large quantities of antimicrobials in animal feeds used in IFAP facilities. While intended to produce ideal market weights and consistency among livestock and poultry, these drugs have been found to spur the mutation of many forms of bacteria and other pathogens into forms that are increasingly resistant to once-reliable antimicrobial and antibiotic drugs. Humans are increasingly exposed to a number of these resistant strains for which commonly used antibiotics are no longer effective.

Several recent and high profile recalls involving E. coli O157:H7, as well as Salmonella enteritidis serve as graphic reminders of the risk of food borne illnesses – risk that are greatly amplified by the scale and methods common to IFAP. All areas of meat, poultry, egg, and dairy production can potentially contribute to zoonotic disease and food contamination, with dire consequences if they do reach human hosts. A 1999 report estimated that E. coli O157:H7 infections caused approximately 73,000 illnesses, leading to over 2,000 hospitalizations and 60 deaths each year in the United States. Animal manure, especially from cattle, is the primary source of these bacteria, and major routes of human infection include consumption of food and water contaminated with animal wastes.

IFAP facilities also cause occupational health impacts. Workers and operators are exposed to toxic dust and gases that may result in temporary, and in some cases chronic, respiratory irritation, including bronchitis, non-allergic asthma-like syndrome, mucous membrane irritation, and non-infectious sinusitis. In addition to dust, other irritants, such as gases, are generated inside the building from decomposition of animal urine and feces. A 1997 study of chronic (non-IFAP or IFAP) occupational exposures to hydrogen sulfide, a byproduct of some animal waste processing methods, found that such exposures might lead to neuropsychiatric abnormalities including impaired balance, hearing, memory, mood, intellectual function, and visual field performance.

Finally, communities surrounding IFAP facilities are also vulnerable to health hazards from air emissions and water pollution. These hazards include respiratory symptoms, disease and functional impairment, as well as neurobehavioral symptoms. Those with weaker immune systems, such as young children and the elderly, are especially vulnerable. One study showed that North Carolina residents who lived in the vicinity of intensive swine operations exhibited higher rates of tension, depression, anger, reduced vigor, fatigue, and confusion than those who did not live near the facility.

Current monitoring systems in IFAP are inadequate to protect the public from the harmful effects of contamination or disease. Animal identification and meat product labeling practices make tracing infections to the source difficult or impossible. Moreover, IFAP may be legally exempt from mandatory health monitoring, disease reporting and surveillance programs in many cases, so IFAP workers who may carry disease-causing organisms usually are not identified.

“While many features of industrial farm animal production have provided plentiful food products to consumers, there are unacceptable health risks associated with IFAP that must be confronted,” said Dr. Michael Blackwell. “The public should not have to pay for high productivity with their health.”

The Pew Commission was convened in 2005 to study the impacts of dramatic changes in animal agriculture in America over the past 40 years. The decline of the family farm and the concentration of the industry into a relative few large corporations has meant greater efficiency and lowered costs for producers. But this shift has also brought environmental, public health, and socioeconomic problems. Today’s event was part of a series of Capitol Hill issue briefings on these risks and challenges that will culminate in the public release on April 29 of a set of recommendations to address them. The PCIFAP’s two-year study encompassed site visits to production facilities across the country, consultation with industry stakeholders, public health, medical, and agriculture experts, public meetings, and peer-reviewed technical reports.

For more information visit the Pew Commission on Industrial Farm Animal Production Web site.
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CDC Confirms Ties to Virus First Discovered in U.S. Pig Factories

For pictures and video see the website

http://www.hsus.org/farm/news/ournews/swine_flu_virus_origin_1998_042909.html

April 30, 2009

Crowded conditions on factory farms create breeding grounds for new viruses. ©iStockphoto

By Michael Greger, M.D.

Factory farming and long-distance live animal transport apparently led to the emergence of the ancestors of the current swine flu threat.

A preliminary analysis of the H1N1 swine flu virus isolated from human cases in California and Texas reveals that six of the eight viral gene segments arose from North American swine flu strains circulating since 1998, when a new strain was first identified on a factory farm in North Carolina.

This analysis, first released by Columbia University’s Center for Computation Biology, has now been reportedly confirmed by researchers at the University of Edinburgh, St. Jude's Children's Research Hospital and virologist Ruben Donis, chief of the molecular virology and vaccines branch at the U.S. Centers for Disease Control and Prevention. Dr. Robert Webster, the director of the U.S. Collaborating Center of the World Health Organization, and considered the "godfather of flu research,"[1] is reported as saying "The triple reassortant in pigs [first discovered in the U.S. in 1998] seems to be the precursor."

Plaguing People and Pigs

The worst plague in human history was triggered by an H1N1 avian flu virus, which jumped the species barrier from birds to humans[2] and went on to kill as many as 50 to 100 million people in the 1918 flu pandemic.[3] No disease, war or famine ever killed so many people in so short a time. We then passed the virus to pigs, where it has continued to circulate, becoming one of the most common causes of respiratory disease on North American pig farms.[4]

For media interviews with Dr. Michael Greger, please contact Liz Bergstrom at ebergstrom@humanesociety.org or 301-258-1455. ©The HSUS

In August 1998, however, a barking cough resounded throughout a North Carolina pig factory in which all the thousands of breeding sows fell ill.[5] A new swine flu virus was discovered on that factory farm, a human-pig hybrid virus that had picked up three human flu genes. By the end of that year, the virus acquired two gene segments from bird flu viruses as well, becoming a never-before-described triple reassortment virus—a hybrid of a human virus, a pig virus, and a bird virus—that triggered outbreaks in Texas, Minnesota, and Iowa.[6]

Within months, the virus had spread throughout the United States. Blood samples taken from 4,382 pigs across 23 states found that 20.5% tested positive for exposure to this triple hybrid swine flu virus by early 1999, including 100% of herds tested in Illinois and Iowa, and 90% in Kansas and Oklahoma.[7] According to the current analysis, it is from this pool of viruses that the current swine flu threat derives three-quarters of its genetic material.[8]

Tracing the Origins of Today's Virus

Since the progenitor of the swine flu virus currently threatening to trigger a human pandemic has now been identified, it is critical to explore what led to its original emergence and spread. Scientists postulate that a human flu virus may have starting circulating in U.S. pig farms as early as 1995, but "by mutation or simply by obtaining a critical density, caused disease in pigs and began to spread rapidly through swine herds in North America. [emphasis added]"[9] It is therefore likely no coincidence that the virus emerged in North Carolina, the home of the nation’s largest pig production operation. North Carolina has the densest pig population in North America and reportedly boasts more than twice as many corporate pig mega-factories as any other state.[10]

The year of emergence, 1998, was the year North Carolina's pig population hit ten million, up from two million just six years earlier.[11] Concurrently, the number of pig farms was decreasing, from 15,000 in 1986 to 3,600 in 2000.[12] How can five times more animals be raised on almost five times fewer farms? By crowding about 25 times more pigs into each operation.

In the 1980s, more than 85% of all North Carolina pig farms had fewer than 100 animals. By the end of the 1990s, operations confining more than 1,000 animals controlled about 99% of the state's pig population.[13] Given that the primary route of swine flu transmission is thought to be the same as human flu—via droplets or aerosols of infected nasal secretions[14]—it's no wonder experts blame overcrowding for the emergence of new flu virus mutants.

Intensive Crowding and Long-Distance Transport

Starting in the early 1990s, the U.S. pig industry restructured itself after Tyson's profitable chicken model of massive industrial-sized units. As a headline in the trade journal National Hog Farmer announced, "Overcrowding Pigs Pays—If It's Managed Properly."[15] The majority of U.S. pig farms now confine more than 5,000 animals each. A veterinary pathologist from the University of Minnesota stated the obvious in Science: "With a group of 5,000 animals, if a novel virus shows up it will have more opportunity to replicate and potentially spread than in a group of 100 pigs on a small farm."[16]

Dr. Robert Webster, one of the world's leading experts of flu virus evolution, blames the emergence of the 1998 virus on the "recently evolving intensive farming practice in the USA, of raising pigs and poultry in adjacent sheds with the same staff," a practice he calls "unsound."[17] North Carolina is also one of the nation's largest poultry producers, slaughtering nearly three-quarters of a billion chickens[18] and confining enough hens to produce nearly 3 billion eggs.[19]

Once the new viral mutant appeared in 1998, the rapid dissemination across the country has been blamed on long-distance live animal transport.[20] In the United States, pigs travel coast to coast. They can be bred in North Carolina, fattened in the corn belt of Iowa, and slaughtered in California.[21] While this may reduce short-term costs for the pork industry, the highly contagious nature of diseases like influenza (perhaps made further infectious by the stresses of transport) needs to be considered when calculating the true cost of long-distance live animal transport.

"A Recipe for Disaster"

The remaining two gene segments of the H1N1 swine flu virus now spreading in human populations around the world appear to come from a swine flu viral lineage circulating in Eurasia, where similar conditions may be to blame. "Influenza [in pigs] is closely correlated with pig density," said a European Commission-funded researcher studying the situation in Europe.[22] As such, Europe's rapidly intensifying pig industry has been described in Science as "a recipe for disaster."[23] Some researchers have speculated that the next pandemic could arise out of "Europe's crowded pig barns."[24] In Europe in 1993, a bird flu virus had adapted to pigs, acquiring a few human flu virus genes and infected two young Dutch children, displaying evidence of limited human-to-human transmission.[25]

The European Commission's agricultural directorate warns that the "concentration of production is giving rise to an increasing risk of disease epidemics."[26] Concern over epidemic disease is so great that Danish laws have capped the number of pigs per farm and put a ceiling on the total number of pigs allowed to be raised in the country.[27]

No such limit exists in the United States or in Mexico. The fact that the first confirmed human case of swine flu appeared in close proximity to the largest pig factory in Mexico, which slaughters nearly a million pigs a year (out of a country-wide total of 15 million), may not have been a coincidence.

Warnings Unheeded

The public health community has been warning about the risks posed by factory farms for years. More than five years ago, in 2003, the American Public Health Association, the largest and oldest association of public health professionals in the world, called for a moratorium on factory farming.[28] In 2005, the United Nations urged that "[g]overnments, local authorities and international agencies need to take a greatly increased role in combating the role of factory-farming," which, they said, combined with live animal markets, "provide ideal conditions for the [influenza] virus to spread and mutate into a more dangerous form."[29]

Last April, the Pew Commission on Industrial Farm Animal Production released its final report. The prestigious, independent panel chaired by a former Kansas Governor and including a former U.S. Secretary of Agriculture, former Assistant Surgeon General, and the Dean of the University of Iowa College of Public Health, concluded that industrialized animal agriculture posed "unacceptable" public health risks: "Due to the large numbers of animals housed in close quarters in typical [industrial farm animal production] facilities there are many opportunities for animals to be infected by several strains of pathogens, leading to increased chance for a strain to emerge that can infect and spread in humans."[30]

Specific to the veal crate-like metal stalls that confine breeding pigs like those on the North Carolina factory from which the first hybrid swine flu virus was discovered in North America, the Pew Commission asserted that "[p]ractices that restrict natural motion, such as sow gestation crates, induce high levels of stress in the animals and threaten their health, which in turn may threaten human health."[31] Unfortunately we don't tend to "shore up the levees" until after the disaster, but now that we know swine flu viruses can evolve to efficiently transmit human-to-human we need to follow the Pew Commission's recommendations to abolish extreme confinement practices like gestation crates as they're already doing in Europe, and to follow the advice of the American Public Health Association to declare a moratorium on factory farms.

A "Reservoir of Viruses" in the U.S.

With massive concentrations of farm animals within whom to mutate, these new swine flu viruses in North America seem to be on an evolutionary fast track, jumping and reassorting between species at an unprecedented rate.[32] This reassorting, Webster's team concludes, makes the 65 million strong U.S. pig population an "increasingly important reservoir of viruses with human pandemic potential."[33] "We used to think that the only important source of genetic change in swine influenza was in Southeast Asia," said Christopher Olsen, a molecular virologist at the University of Wisconsin, Madison. Now, "we need to look in our own backyard for where the next pandemic may appear."[34]

Dr. Michael Greger is director of public health and animal agriculture for The Humane Society of the United States.

References

[1] Council on Foreign Relations. 2005. Session 1: Avian flu-where do we stand? Conference on the Global Threat of Pandemic Influenza, November 16. http://cfr.org/publication/9230/council_...uenza_session_1.html.

[2] Belshe RB. 2005. The origins of pandemic influenza-lessons from the 1918 virus. New England Journal of Medicine 353(21):2209-11.

[3] Johnson NPAS, Mueller J. Updating the accounts: global mortality of the 1918–1920 "Spanish" influenza pandemic. Bull Hist Med. 2002;76:105–15.

[4] Zhou NN, Senne DA, Landgraf JS, et al. 1999. Genetic reassortment of avian, swine, and human influenza A viruses in American pigs. Journal of Virology 73:8851-6. http://birdflubook.org/resources/ZHOU8851.pdf.

[5] Wuethrich B. 2003. Chasing the fickle swine flu. Science 299:1502-5. http://birdflubook.org/resources/WUETHRICH1502.pdf.

[6] Zhou NN, Senne DA, Landgraf JS, et al. 1999. Genetic reassortment of avian, swine, and human influenza A viruses in American pigs. Journal of Virology 73:8851-6. http://birdflubook.org/resources/ZHOU8851.pdf.

[7] Webby RJ, Swenson SL, Krauss SL, Gerrish PJ, Goyal SM, and Webster RG. 2000. Evolution of swine H3N2 influenza viruses in the United States. Journal of Virology 74:8243-51.

[8] Rabadan, R. 2009. Influenza A (H1N1) "swine flu": worldwide (04) [1] ProMED Digest 2009. 28 April. Volume 2009 : Number 196. http://www.promedmail.org/pls/otn/f?p=2400:1001:1580522401053605::NO::F2400_P1001_BACK_PAGE,F2400_P1001_
PUB_MAIL_ID:1000,77250.

[9] Webby RJ, Swenson SL, Krauss SL, Gerrish PJ, Goyal SM, and Webster RG. 2000. Evolution of swine H3N2 influenza viruses in the United States. Journal of Virology 74:8243-51.

[10] Environmental Defense. 2000. Factory hog farming: the big picture. November. http://www.edf.org/documents/2563_FactoryHogFarmingBigPicture.pdf.

[11] Duke University Center on Globalization, Governance and Competitiveness. 2006. Hog farming overview. February 23. http://www.soc.duke.edu/NC_GlobalEconomy/hog/overview.php.

[12] North Carolina Department of Agriculture and Consumer Services. 2001. North Carolina agriculture overview. February 23. http://ncagr.com/stats/general/livestoc.htm.

[13] Wuethrich B. 2003. Chasing the fickle swine flu. Science 299:1502-5. http://BirdFluBook.org/resources/WUETHRICH1502.pdf.

[14] Brown IH. 2000. The epidemiology and evolution of influenza viruses in pigs. Veterinary Medicine 74:29-46. http://BirdFluBook.org/resources/Brown29.pdf.

[15] 1993. Overcrowding pigs pays-if it's managed properly. National Hog Farmer, November 15.

[16] Wuethrich B. 2003. Chasing the fickle swine flu. Science 299:1502-5. http://BirdFluBook.org/resources/WUETHRICH1502.pdf.

[17] Webster RG and Hulse DJ. 2004. Microbial adaptation and change: avian influenza. Revue Scientifique et Technique 23(2):453-65.

[18] USDA. 2009. Poultry Slaughter 2008. Annual Summary. http://usda.mannlib.cornell.edu/usda/current/PoulSlauSu/PoulSlauSu-02-25-2009.pdf

[19] USDA. 2009. Chickens and Eggs 2008 Summary. http://usda.mannlib.cornell.edu/usda/current/ChickEgg/ChickEgg-02-26-2009.pdf

[20] Wuethrich B. 2003. Chasing the fickle swine flu. Science 299:1502-5. http://birdflubook.org/resources/WUETHRICH1502.pdf.

[21] Shields DA and Mathews KH Jr. 2003. Interstate livestock movements. USDA Economic Research Service: Electronic Outlook Report from the Economic Research Service, June. usda.mannlib.cornell.edu/reports/erssor/livestock/ldp-mbb/2003/ldp-m108-01.pdf.

[22] MacKenzie D. 1998. This little piggy fell ill. New Scientist, September 12.

[23] Ibid.

[24] Delgado C, Rosegrant M, Steinfeld H, Ehui S, and Courbois C. 1999. Livestock to 2020: the next food revolution. Food, Agriculture, and the Environment Discussion Paper 28. For the International Food Policy Research Institute, the Food and Agriculture Organization of the United Nations and the International Livestock Research Institute. http://ifpri.org/2020/dp/dp28.pdf.

[25] Webster RG, Sharp GB, and Claas CJ. 1995. Interspecies transmission of influenza viruses. Americal Journal of Respiratory and Critical Care Medicine 152:525-30.

[26] MacKenzie D. 1998. This little piggy fell ill. New Scientist, September 12, p. 1818.

[27] Ibid.

[28] American Public Health Association. 2003. Precautionary moratorium on new concentrated animal feed operations. Policy number 20037. www.apha.org/advocacy/policy/policysearch/default.htm?id=1243.

[29] United Nations. 2005. UN task forces battle misconceptions of avian flu, mount Indonesian campaign. UN News Centre, October 24. un.org/apps/news/story.asp?NewsID=16342&Cr=bird&Cr1=flu

[30] Pew Commission on Industrial Farm Animal Production. 2008. Expert panel highlights serious public health threats from industrial animal agriculture. Press release issued April 11. www.pewtrusts.org/news_room_detail.aspx?id=37968. Accessed August 26, 2008.

[31] Pew Commission on Industrial Farm Animal Production. 2008. Putting meat on the table: industrial farm animal production in America. Executive summary, p. 13. www.ncifap.org/_images/PCIFAPSmry.pdf. Accessed August 26, 2008.

[32] Wuethrich B. 2003. Chasing the fickle swine flu. Science 299:1502-5. http://birdflubook.org/resources/WUETHRICH1502.pdf.

[33] Webby RJ, Rossow K, Erickson G, Sims Y, and Webster R. 2004. Multiple lineages of antigenically and genetically diverse influenza A virus co-circulate in the United States swine population. Virus Research 103:67-73. http://BirdFluBook.org/resources/webby67.pdf.

[34] Wuethrich B. 2003. Chasing the fickle swine flu. Science 299:1502-5. http://BirdFluBook.org/resources/WUETHRICH1502.pdf.

Barrick and Argentine Officials Violently Assault Women at Roadblock

Barrick and Argentine Officials Violently Assault Women at Roadblock : Intercontinental Cry

On April 14, a group of Argentine government officials and employees of Barrick Gold Corporation, carried out a violent assault against Women at the Famatina mining camp in the province of La Rioja, where a road blockade has stood for the past two years.

When the officials arrived, a group of Women from the “Self-Organized (Autoconvocados) Neighbors of Famatina for Life,” gathered at site and lowered a metal bar they installed to deny the company’s passage to the mine site.

The officials and Barrick employees then began to ram their trucks against the barrier, but “without any success,” explains an April 14 media alert.

The officials then exited their vehicles and carried out a violent assault against a handful of women, who had peacefully sat down in front the vehicles - first shoving them, and then kicking and striking the women with their fists.

Plight? What Plight? NYT Shoddy Journal on Multiracials

This is from the Mulatto Diaries by Tiffany Jones:

"My jaw dropped.
Then I wrote this…
Mr. Levitt,
I was deeply offended by your “Plight of Mixed-Race Children” blog post. Clearly the “tragic mulatto” was already a joke to you when you set out to determine how the young ones are doing. First of all, the “more attractive” stereotype is becoming as irritating to me as the blacks and fried chicken thing. If you took this seriously you would look beyond that to find out if those “attractive” children perceived themselves that way. Or did they feel physically flawed somehow because they did not fit in easily with either their black or white counterparts. Did their peers regard them as attractive? I believe that this more attractive thing comes in to play after adolescence and should have had no place in your “study”.
You also seem to believe that all of these young black and white people have white mothers and black fathers. Black fathers being absent and poor, generally leaving white mothers on their own to care for their children in lower class surroundings. That’s what I infer from this crappy post. Come the f*** on! Many biracials have a white father and a black mother. Did the possibility of that even cross your mind? I am left to assume that this white man, Steven D. Levitt, wouldn’t dream of procreating with a black woman and that you judge the white men who would as somewhat less than yourself.
Where on Earth did you get your “data”? What questions exactly were you trying to answer? Why not interview biracial adults about their adolescent experience? Why do you even care what it’s like to grow up black and white? Clearly you do not.
I am 32. I have a black mother and a white father. I grew up in the wealthy suburbs of Detroit. I excelled academically and artistically. I earned a full-ride to the University of Michigan without trying very hard. During my adolescent years I did not have sex, a cigarette, a sip of alcohol, or get into a fight. I did not come away from my adolescent experience thinking “That was kind of rough, but as least I am more attractive than all those ‘monoracial’ kids.” I have a vlog on youtube called “mulatto diaries”. Should you someday find that you truly care to know what it is like to grow up in America with a black and a white parent, watch it. You will find the stories of many other biracial people there. You don’t have to take only my word for it.
fyi: I’ll be posting this on my blog mulattodiaries.wordpress.com.
Sincerely,Tiffany Jones"

Why ignore pre-1960's mixing as well, Mr. Leavitt? Your ignorance is showing! Thank you, Tiffany, for exposing his willful ignorance.

What Tami Said: Between "Mammy" and "Miss Ann"-- The "problem" with Michelle

What Tami Said: Between "Mammy" and "Miss Ann"-- The "problem" with Michelle

Excellent essay on how American society view our First Lady Michelle Obama. As I wrote yesterday, there are haters of Michelle Obama out there.

Conservative Blogger's Hatefest of Michelle Obama

What's wrong with wanting to look glamourous? Plain old jealousy and resentment.
Michelle Obama is beautiful, inside and out. Their kids, Sasha and Malia, look great too

Mind you, this is not surprising, coming from a man with anti-Black views. Here's a racist/sexist commentary from a conservative blogger regarding Black women he wrote in 2002. He complained that there are too much Black women in the top 100 women of 2002 and not enough Asian women. He also complained about bw/wm IR.

His views mirrors David Yeagley's. I believe David copied his views on multiracial/black beauty from him. He basically wrote that Black women aren't attractive, even multiracial beauties such as Halle Berry. He also wondered why are so many White men are pursuing Black women as if it's not normal. Why should he care?

BLACK CHICKS: I was perusing FHM’s list of the 100 Sexiest Women in the World 2002 in search of a story—the things I do for my readers—when I noticed how white they are. According to our race-obsessed Census Bureau, over 12 percent of Americans are black, but the mostly-US readership of FHM selected only 8 black women:

#3: Halle Berry—Actress #21: Beyoncé Knowles—Singer #23: Tyra Banks—Model #34: Janet Jackson—Singer #36: Mariah Carey—Singer, Weirdo #80: Alicia Keys—Singer #82: Aaliyah—Singer, Corpse (alas) #85: Samantha Mumba—Singer

Even the above list is a little misleading, because a quick visit to MixedFolks.com revealed that four of these women (Halle, Mariah, Alicia, and Samantha) are decidedly biracial, having one African and one European parent. I use the phrase decidedly biracial after having seen this article that discusses DNA testing.

A study of the genetic makeup of 3,000 Americans reveals how racially mixed we really are:

Among self-identified whites in Shriver’s sample, the average black admixture is only 0.7 percent. That’s the equivalent of having among your 128 great-great-great-great-great-grandparents (who lived around two centuries ago), 127 whites and one black.

It appears that 70 percent of whites have no African ancestors. Among the 30 percent who do, the black admixture is around 2.3 percent, which would be like having about three black ancestors out of those 128.

In contrast, African-Americans are much more racially mixed than European-Americans. Yet, Shriver’s study shows that they are less European that was previously believed.

Earlier, cruder studies, done before direct genetic testing was feasible, suggested that African-Americans were 25 or even 30 percent white. Shriver’s project is not complete, but with data from 25 sites already in, he is coming up with 17-18 percent white ancestry among African-Americans. That’s the equivalent of 106 of those 128 of your ancestors from seven generations ago having been Africans and 22 Europeans.

According to Shriver, only about 10 percent of African-Americans are over 50 percent white.

Of course, another way of looking at these data is that a limited amount of black ancestry is enough to make you “look black” from the standpoint of identification. The “one drop” rule may have been ugly from a social and legal perspective, but perhaps reflected a practical reality.

Getting back to our list: Does the mixing of races create an exotic look that has helped these four women? Perhaps there’s a “best of both worlds” effect that helps set these women apart. But why the shortage of black women overall? One simple explanation would be that the readership of FHM is skewed towards white men and their standards of beauty are rooted in the familiar. But maybe it’s because black women just aren’t as pretty.

Consider another piece written by the author of the DNA article above, Steve Sailer, discussing why interracial marriage shortchanges black women and Asian men. He posits some universal standards of beauty that leaves black women behind because of their physiognomy:

By emphasizing how society encourages us to marry people like ourselves, sociologists miss half the picture: by definition, heterosexual attraction thrives on differences ... Opposites attract. And certain race/sex pairings seem to be more opposite than others. The force driving these skewed husband - wife proportions appears to be differences in perceived sexual attractiveness.

On average, black men tend to appear slightly more and Asian men slightly less masculine than white men, while Asian women are typically seen as slightly more and black women as slightly less feminine than white women. Sailer specifically cites height, hair length, and muscularity as disadvantages for black women—and advantages for Asian women.

The only problem with this theory in our case? The list of 100 contains only two Asian women: Lucy Liu (#19) and Zhang Ziyi (#91).

WHITE GUYS & BLACK CHICKS: The taboo against black women dating white men is breaking down. The growing education gap between black men and women and the cold fact that many black men are incarcerated or murder victims are causal factors. (By using education as a surrogate for wealth, this article skirts the issue that white men are wealthier than black men.

We can’t have black women looking like gold diggers, can we?) One question the article doesn’t fully confront is why white men are going after dark meat, though it acknowledges the “exotic look” factor I attributed to biracial women in a previous post. Black guys still have their fans, though.

Mickey Kaus reports: Then there is the final taboo issue—black male sexuality. Fear of black male sexuality was the psychological engine of Jim Crow, Nicholas Lemann has argued. And respect for it is the unwritten explanation for a lot of the resistance to cross-race dating (between 1960 and 1980, for example). I once asked a multi-degreed, accomplished black woman friend of mine (who dated whites) why so many other black women restricted their “options” to black men. She paused thoughtfully, stroked her chin, and said: “I guess it’s the sex.”

That's disgusting and racist to reduce Black men to sexual objects, Black women as gold diggers, and White men as fetishist who date/marry Black women. Can people find black women and white men loving each other instead of imputing some motive to it? The same goes for black men and women relationships.

Glitter 1/12 -Mariah carey

Turkish Anchor Reports On Obama In Blackface (VIDEO)

Turkish Anchor Reports On Obama In Blackface (VIDEO): "A Turkish news anchor wore dark face paint while he reported on President Obama's trip to the country."

Welcome to Turkey and Spirit of Ancestry

Afrosphere

Hat tip to Runoko Rashidi and TheBlackList

ISTANBUL - The black population of Turkey has a message for U.S. President Barack Obama.

Mustafa Olpak, author, activist and president of the Africans’ Culture and Solidarity Foundation in Izmir and other foundation members crave a meeting with Obama Ğ even for just a few minutes. But with Obama’s schedule and the foundation’s lack of funds for the trip, the group realizes it will not happen. Still, Olpak told Hürriyet Daily News & Economic Review they were excited about the visit. “The spirits of his African ancestors, who were enslaved and excluded from the white man’s world, are with Obama,” he said.

Black people were not being bought and sold only in Europe and America, but in the Ottoman Empire, too. The island of Crete in the Aegean Sea was the most important center for the slave trade. Olpak comes from a family whose members were sold as slaves on this island. Although the family chose to remain silent on the matter, Opak has broken the silence with his recent book “Kölekıyısı-Kenya’dan Istanbul’a” (Slave Shore Ğ from Kenya to Istanbul). The book, out on Punto Publishing, features photographs and documents. There was also an exhibit at the EU center in Ankara, which opened with the book’s release.

The exhibit, titled “The Blacks of Turkey,” will be reopened in Ankara on the occasion of Obama’s visit with additional photographs from Opak’s family album added. “Maybe Obama cannot visit the foundation to meet us but he should see the exhibit at least,” said Olpak.

AP Says It's Going To Sue Aggregators

Techdirt

Given some of the Associated Press's recent actions, this won't come as a surprise, but the AP has now announced that it will start suing any news aggregator that doesn't share its profits with the AP:

"We can no longer stand by and watch others walk off with our work under misguided legal theories."

I'm a bit curious what those "misguided theories" are... because copyright law and rules concerning fair use seem pretty clear, and search engines aggregating info and sending people to your site has been ruled fair use before. So, perhaps the AP chairman is talking about some other "misguided" legal theory? Another AP person claims: "This is not about defining fair use. There's a bigger economic issue at stake here that we're trying to tackle." But she neglects to say what that is, other than our old business model sucks, and we've got no freaking clue how to adapt to the changing market place, so this is the best we've got...

Whenua Fenua Enua Vanua: G20 Meltdown Solidarity

Whenua Fenua Enua Vanua: G20 Meltdown Solidarity: "Wednesday April 1st

12-12.30 pm

British Consulate
Melbourne
90 Collins Street


'People are in an incendiary mood,' says Bone. '1 April will see the biggest ructions on the street since the poll-tax riots and possibly even the Gordon riots of 1780. I don't think politicians realise quite how angry we are. In the past six months, this country has been turned upside-down. A deep recession has been created by a few greedy bankers and as a result, thousands have lost their homes and jobs. A dam of resentment has built up and 1 April is when all these pissed-off people march on the City to take what's theirs. Capitalism itself is on the ropes.'



http://www.g-20meltdown.org/
http://www.indymedia.org.uk/en/2009/03/425026.html"

Attorney Chiquita Tate Murdered

Slain attorney Chiquita Tate was such a believer in the legal system that she had a tattoo of Lady Justice on her back, college friend T. J. Crawford recalled.

Chiquita Tate was in the midde of a high-profile murder trial when she was stabbed to death in her law office.

"She just had an attachment to justice and doing what's right by people. She was always very serious about that," said Crawford, a teacher and community organizer in Chicago, Illinois.
But Tate, described by colleagues as a tenacious defense attorney who fought for her clients, could not save herself.
Family members and friends in Chicago; Atlanta, Georgia, and Tate's adopted home -- tiny Baker, Louisiana -- are reeling from the grisly details of Tate's slaying, and police say it was at the hands of her husband, Greg Harris. They had been married about 14½ months.
Harris, 37, is in custody, accused of stabbing Tate to death. He is charged with second-degree murder and the illegal use of a dangerous weapon. A judge last week set his bond at $500,000.
In a phone interview with CNN, Harris' attorney, Lewis Unglesby, said police have the wrong man.

"Greg Harris by all accounts ... is innocent. I don't know anybody that thinks he did it, except the police," Uglesby said. "There's nothing in his background. He has cooperated completely with the police; he's signed everything they've asked him to sign. He's let them search his house, his car."
Tate, 34, had started her own law firm in downtown Baton Rouge, Louisiana, and was doing well, family and acquaintances said.

"She was up and coming," said Cpl. L'Jean McKneely, a police spokesman in Baton Rouge.
Tate was representing a defendant in a high-profile murder trial when, police say, she became a homicide victim herself. Her body was found inside her law office on February 20. Tate was stabbed 38 times, according to a police warrant for Harris' arrest obtained by CNN.
While questioning Harris about Tate's death, police discovered an outstanding warrant for him in connection with a battery-domestic violence case. It stemmed from a December 22, 2007, incident at the couple's home.

Harris was accused of using "force and violence" against Tate, according to Baker City Court records. The court said Harris entered a not guilty plea on March 6, 2008, but did not appear for a May 8 pretrial conference. A warrant was issued for his arrest for contempt of court.

Police Shooting in Pasadena - NAM

NAM

Black residents in Pasadena are furious with the Pasadena Police Department after the shooting death of Leroy Barnes, 38. Adena Cole, a friend of Barnes' family, is fearful for members of her community.

Barnes was riding in the backseat of a car driven by Ameka Edwards at 4:20 p.m. on February 19 when they were pulled over by the police. It is unclear exactly what led to the fatal shooting of Barnes because the Pasadena Police Department has put a hold on the autopsy results, written and taped reports, recorded interviews regarding the shooting and surveillance videos from the officers squad car.

The first report from the Pasadena Police Department was that Barnes stepped out of the car and fired upon the officers. But a report the next day from the police department said that Barnes did not step out of the car and that he did not fire a shot.

Barnes does have a criminal record, and there was a gun in the car, but according to Cole, he did not have the gun in his possession at the time of the shooting. She claims that when the car was pulled over Edwards told Barnes to put the gun into her purse because she is a security officer and has a license to carry a gun.